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氧化应激与糖尿病肾病 被引量:15

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摘要 糖尿病肾病是糖尿病严重的并发症,至今其发病机制尚未完全阐明,目前认为氧化应激可能起重要作用。本文就氧化应激与糖尿病肾病的关系做一综述。
出处 《中国实用医药》 2008年第3期138-139,共2页 China Practical Medicine
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  • 1李才,邓义斌,王丽娟,张秀云,邹雅斌,侯芳玉.实验性糖尿病大鼠肾脏自由基防御机能的改变及其意义[J].中国病理生理杂志,1995,11(6):638-641. 被引量:32
  • 2[2]Maritim AC,Sanders RA,Watkins JB.Diabetes,ox-idative stress,and antioxidants:a review.J Biochem Mol Toxicol,2003,17:24-38.
  • 3[3]Brownlee M.Biochemistry and molecular cell biology of diabetic comp lications.Nature,2001,414:813-820.
  • 4[4]Xu Y,Osborne BW,Stanton RC.Diabetes Causes Inhibition of Glucose6-Phosphate Dehydrogenase via Activation of Protein Kinase A which Contributes to Oxidative Stress in Rat Kidney Cortex.Am J Physiol Renal Physiol,2005,289 (5):1040-1047.
  • 5陈玲,贾汝汉,丁国华,刘红燕,杨定平,彭隽,田少江.缬草油对2型糖尿病大鼠肾脏的保护作用及其机制探讨[J].中华肾脏病杂志,2003,19(3):168-172. 被引量:42
  • 6[6]Aksun AS,Ozmen B,Ozmen D,et al.Serum and urinary nitric oxide in Type 2 diabetes with or without microalbuminuria:relation to glomerular hyperfiltration.J Diabetes Complications,2003,17 (6):343-348.
  • 7[7]Bands N,Nakamura T,Matsumura M,et al.Possible relationship of monocyte chemoattratant protein-1 with diabetic nephropathy.Kinney Int,2000,58 (2):684-686.
  • 8[8]Scivittaro V,Ganz MB,Weiss MF.AGEs induce oxidative stress and activate protein kinase C-beta(Ⅱ) in neotal mesangial cells.Am J Physiol Renal Physiol,2000,278 (4):676-678.
  • 9[9]Susztak K,Raff AC,Schiffer M,et al.Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy.Diabetes,2006,55 (1):225-233.
  • 10[10]Ha H,Yu MR,Choi YJ,et al.Role of high glucose-induced nuclear factor-kappaB activation in monocyte chemoattractant protein-1 expression by mesangial cells.J Am Soc Nephrol,2002,13 (4):894-902.

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