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丙型肝炎病毒与JAK-STAT信号转导系统 被引量:12

Hepatitis C virus and signal transduction system of JAK-STAT
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摘要 0引言 丙型肝炎病毒(HCV)感染具有显著的慢性化倾向,而且只有部分患者对于干扰素α(IFN α)的治疗有较好的应答.为什么会存在这种情况和差别,这是与HCV-肝细胞之间相互作用的分子生物学机制和基础来决定的[1].
出处 《世界华人消化杂志》 CAS 2003年第4期464-466,共3页 World Chinese Journal of Digestology
基金 国家自然科学基金 No.C39970674 C03011402
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参考文献19

  • 1成军.慢性病毒性肝炎发病机制的分子生物学研究[J].世界华人消化杂志,2002,10(2):125-128. 被引量:141
  • 2成军.丙型肝炎病毒感染慢性化的分子生物学机制[J].国外医学(病毒学分册),2000,7(1):29-32. 被引量:33
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二级参考文献72

共引文献161

同被引文献141

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