摘要
目的:探讨纤维蛋白原(FIB)、同型半胱氨酸(HCY)、尿酸(UA)与维医证型的相关性。方法:将200例冠心病患者进行辨证分型,检测FIB,HCY和UA。结果:(1)在西医分型中FIB、HCY、UA水平从高到低顺序为:不稳定型心绞痛>稳定型心绞痛。(2)异常黑胆质组FIB水平明显高于非异常黑胆质组(P<0.05)。异常血液质组HCY、UA水平明显高于非异常血液质组(P<0.05)。(3)异常黑胆质组心电图ST异常阳性率明显高于非异常黑胆质组(P<0.05)。结论:冠心病维医异常黑胆质型与FIB的关系最密切,而异常血液质型与HCY、UA的关系最密切,可作为反映冠心病异常黑胆质型、异常血液质型的客观依据。
Objective: To explore fibrinogen, homocysteine, uric acid level of the patients with coronary heart disease and the correlation between the traditional Uyghur medicine syndrome type. Methods: Grouped the 200 cases of coronary heart disease patients with uyghur medicine syndrome type, levels of fibrinogen, homocysteine, uric acid were determined. Results:(1)In western medicin classifications, the level of plasma FIB, HCY, UA in unstable angina group were higher than that in stableangina(SA) group.(2)The level of FIB in abnormal savda group were significantly higher than non-abnormal savda group(P<0.05). The level of HCY, UA in abnormal khan group were higher than non-abnormal khan group(P<0.05).(3)ECG ST abnormalitys in abnormal savda group was significantly higher than that in non-abnormal savda group(P<0.05). Conclution: Abnormal savda and abnormal khan are the brief pathogenic factors, and the abnormal belghem is principal syndrome. The abnormal savda type of CHD is most closely to the FIB, and abnormal khan type is mostly closely to the HCY, UA, that can be used as reflected CHD abnormal savda and blood syndromes of the objective indicators. The abnormal savda patients in every syndrome type must to pay attention to the possibility of myocardial ischemia at any time. Throw relationship study between traditional Uyghur medicine abnormal hilit differentiation of coronary heart disease patients and FIB, HCY, UA, as well as coronary heart disease traditional Uyghur medicine pathogenesis of the objective of the research to find new clues.
出处
《中华中医药杂志》
CAS
CSCD
北大核心
2017年第10期4664-4666,共3页
China Journal of Traditional Chinese Medicine and Pharmacy
关键词
冠心病
维吾尔医证型
纤维蛋白原
同型半胱氨酸
尿酸
Coronary heart disease
Uyghur medicine syndrome type
Fibrinogen
Homocysteine
Uric acid
