摘要
陷窝蛋白-1作为陷窝的标志蛋白，通过其脚手架结构域寡聚许多细胞信号转导分子，参与许多病理生理过程，也通过不同的途径调控脑缺血后炎症反应。文章对近年来陷窝蛋白-1与缺血性卒中后炎性反应相关研究进展进行了回顾，重点讨论其炎症调控机制。
As a cavola marker protein, caveolin-1 participates in many pathophysiological processes through its scaffolding domain oligomering many celular signal transduction molecules, and also regulates inflammation after cerebral ischemia through different pathways. This article reviews advances in caveolin-1 and inflammation after ischemic stroke in recent years, mainly focusing on its mechanism in regulating inflammation.
出处
《国际脑血管病杂志》
2016年第3期259-262,共4页
International Journal of Cerebrovascular Diseases
基金
国家自然科学基金，湖南省自然科学基金(2015JJ2105) Fund programs:National Natural Science Foundation of China，Natural Science Foundation of Hunan Province of China
作者简介
通信作者：刘柏炎，Email：liubaiyan@126．com
