摘要
A model of incomplete cerebral ischemia caused by bilateral ligation of the commoncarotid arteries in rats was used to examine the effect of naomaikang on the relief of cellulardamage following cerbral ischemia.Cellular damage and the formation of peroxidation productswere reduced in rats pretreated with naomaikang.It has been suggested that the free radical re-action involved with tissue damage,particularly with the injury of neurons in cerebral ischemia.The results showed that the degree of lipid peroxidation can be lowered by the pretreatmentwith naomaikang in the model of incomplete cerebral ischemia,and suggested that this drug isbeneficial to the recovery of cerebral ischemia in humans.
A model of incomplete cerebral ischemia caused by bilateral ligation of the common carotid arteries in rats was used to examine the effect of naomaikang on the relief of cellular damage following cerbral ischemia.Cellular damage and the formation of peroxidation products were reduced in rats pretreated with naomaikang.It has been suggested that the free radical re- action involved with tissue damage,particularly with the injury of neurons in cerebral ischemia. The results showed that the degree of lipid peroxidation can be lowered by the pretreatment with naomaikang in the model of incomplete cerebral ischemia,and suggested that this drug is beneficial to the recovery of cerebral ischemia in humans.
