8Mayinger B,Honer P,Jordan M,et al.Light-induced autofluorescence spectroscopy for the endoscopic detection of esophageal cancer.Gastrointest Endosc,2001,54:195.
10FagiIndes RB,Mello CR,Tellns P,et al.P53 protein in esophageal mucosa of individuals at high risk of squamous cell carcinoma of the esophagus.Dis Esophagus,2001,14:185-190.
3[1]Viallard JF,Lacombe F,Belloc F, et al. Molecular mechanisms controlling the cell cycle:fundamental aspects and implications for oncology [J].Cancer Radiother, 2001,5(2):109-129.
4[2]Sharpless NE, Bardeesy N, Lee KH, et al. Loss of pl6ink4a with retention of p16 Arf predisposes mice to tumorigenesis [J]. Nature, 2001,4(13):86-91.
5[3]Soussi T. The p53 tumor suppressor gene: from mo-lecular biology to clinical investigation [J]. Ann NY Acad Sci, 2000,9(10):121-137.
6[4]Hussain SP, Harris CC. p53 mutation spectrum and load: The generation of hypotheses linking the expose of endogenous or exogenous carcinogens to human cancer [J]. Mutation Res, 1999,4(28):23-31.
7[5]Wadih A, Ryo N, Frank B, et al. Replacement of the p16/CDKN2 gene suppresses human glioma cell growth [J]. Cancer Res, 1995,5(5):1351-1357.
8[6]Zhou X, Tarmin I, Yin J, et al. The MTS1 gene is freqently mutated in squamouse cell carcinoma of the esophagus human esophageal tumors [J]. Oncogene, 1994,9(12):37-41.
9[7]Liu Q, Yan YX, Mcclure M, et al. MTS1(CDKN2) tumor suppressor gene deletions are a frequent event in esophagus sqamous cancer and pancreatic adenocarcinoma cell lines [J]. Oncogene, 1995,10(3):619-625.
10[8]Mori T, Miura K, Aoki T, et al. Frequent somatic mutation of the MTS1/CDK4(multiple tumor suppressor/cyclin-depen kinase-4 inhibitor) gene in esophageal squamous cell carcinoma [J]. Cancer Res, 1994,54(11):3396-.
