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银屑病发病机制研究中若干问题的思考 被引量：104

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作者 郑敏
机构地区 浙江大学医学院附属第二医院皮肤科
出处 《中华皮肤科杂志》 CAS CSCD 北大核心 2006年第3期121-123,共3页 Chinese Journal of Dermatology
基金 国家自然科学基金资助项目(30471565)
关键词 发病机制研究 银屑病 慢性炎症性增生性皮肤病 干扰素(IFN)-Γ TH1/TH2 角质形成细胞 多基因遗传 T细胞 免疫异常 环境因素
分类号 R758.63 [医药卫生—皮肤病学与性病学]
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参考文献20

  • 1Bos JD,de Rie MA,Teunissen MB,et al.Psoriasis:dysregulation of innate immunity.Br J Dermatol,2005,152:1098-1107.
  • 2郑敏,浙江大学医学院第二医院皮肤科.银屑病是细胞免疫介导性疾病?[J].中华皮肤科杂志,2002,35(2):85-87. 被引量：90
  • 3Sano S,Chan KS,Carbajal S,et al.Stat3 links activated keratinocytes and immunocytes required for development of psoriasis in a novel transgenic mouse model.Nat Med,2005,11:43-49.
  • 4Pittelkow MR.Psoriasis:more than skin deep.Nat Med,2005,11:17-18.
  • 5Bowcock AM,Krueger JG.Getting under the skin:the immunogenetics of psoriasis.Nat Rev Immunol,2005,5:699-711.
  • 6Majewski S,Jablonska S.Possible involvement of epidermodysplasia verruciformis human papillomaviruses in the immunopathogenesis of psoriasis:a proposed hypothesis.Exp Dermatol,2003,12:721-728.
  • 7Moles JP,Tesniere A,Guilhou JJ.A new endogenous retroviral sequence is expressed in skin of patients with psoriasis.Br J Dermatol,2005,153:83-89.
  • 8Xia YP,Li B,Hylton D,et al.Transgenic delivery of VEGF to mouse skin leads to an inflammatory condition resembling human psoriasis.Blood,2003,102:161-168.
  • 9Young HS,Summers AM,Bhushan M,et al.Single-nucleotide polymorphisms of vascular endothelial growth factor in psoriasis of early onset.J Invest Dermatol,2004,122:209-215.
  • 10Zheng M,Zhu F.Overexpression of vascular endothelian growth factor receptors in epidermis of psoriasis vulgaris.J Invest Dermatol,2004,122:862(A1).

二级参考文献47

  • 1邵长庚.我国银屑病的流行和防治现状[J].中华皮肤科杂志,1996,29(2):75-76. 被引量：72
  • 2全国银屑病流行调查组.全国1984年银屑病流行调查报告[J].中华皮肤科杂志,1986,19:253-261.
  • 3Krueger GG, Duvic M. Epidemiology of psoriasis: clinical issues. J Invest Dermatol, 1994, 102: 14S-18S.
  • 4Kruglyak L, Daly M J, Reeve-Daly MP, et al. Parametric and nonparametric linkage analysis: a unified multipoint approach. Am J Hum Genet, 1996, 58: 1347-1363.
  • 5ander E, Kruglyak L. Genetic dissection of complex traits: guidelines for interpreting and reporting linkage results. Nat Genet, 1995,11: 241-247.
  • 6Trembath RC, Clough RL, Rosbotham JL, et al. Identification of a major susceptibility locus on chromosome 6p and evidence for further disease loci revealed by a two stage genome-wide search in psoriasis. Hum Mol Genet, 1997, 6: 813-820.
  • 7Balendran N, Clough RL, Arguello JR, et al. Characterization of the major susceptibility region for psoriasis at chromosome 6p21.3. J Invest Dermatol, 1999, 113: 322-328.
  • 8Oka A, Tamiya G, Tomizawa M,et al.Association analysis using refined microsatellite markers localizes a susceptibility locus for psoriasis vulgaris within a 111 kb segment telomeric to the HLA-C gene. Hum Mol Genet, 1999, 8: 2165-2170.
  • 9Nair RP, Stuart P, Henseler T,et al. Localization of psoriasis-susceptibility locus PSORS1 to a 60-kb interval telomeric to HLA-C.Am J Hum Genet, 2000, 66: 1833-1844.
  • 10Zhang X, Wang H, Te-Shao H, et al. The genetic epidemiology of psoriasis vulgaris in Chinese Han. Int J Dermatol, 2002, 41:663-669.

共引文献201

同被引文献716

引证文献104

二级引证文献396

中华皮肤科杂志
2006年 第3期

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