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白癜风氧化应激发病机制的研究进展 被引量：19

Update of the oxidative stress in the pathogenesis of vitiligo
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摘要 白癜风发病机制复杂,涉及遗传、免疫、氧化应激、功能性黑素细胞凋亡和(或)丢失、神经体液等假说。本文将对Keap1/Nrf2/ARE信号转导通路与氧化应激进行综述。 Pathogenesis of vitiligo is complex,involving genetic,immunity,oxidative stress,apoptosis of functional melanocytes and（ or） loss and neurohormonal hypothesis, etc. Keap1/Nrf2/ARE pathway associated with oxidative stress was reviewed in this paper.
作者 赵晓菲 刘国艳
机构地区 潍坊医学院
出处 《中国麻风皮肤病杂志》 2016年第3期189-191,共3页 China Journal of Leprosy and Skin Diseases
基金 山东省自然科学基金(编号:ZR2013HL068) 潍坊医学院附属医院科技创新基金项目(编号:K12CX1009)
关键词 白癜风 氧化应激 Keap1/Nrf2/ARE信号通路 vitiligo oxidative stress Keap1/Nrf2/ARE signal pathway
分类号 R758.41 [医药卫生—皮肤病学与性病学]
作者简介 通信作者：刘国艳，E-mail：wfliuguoyan@126．com
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  • 1ZENG Jian-guo1,3,HU Yi-xiu2,ZHANG Xiang-qian1 1.Natural Product Research Center,Hunan Agricultural University,Changsha 410016,China 2.Toxicological Laboratory,Hunan Provincial Center for Disease Control and Prevention,Changsha 410005,China 3.Hunan Engineering Research Center of Botanical Extract,Changsha 410331,China.Toxicological Assessment of Trans-resveratrol[J].Chinese Herbal Medicines,2010,2(1):30-40. 被引量：4
  • 2WANG Chang-yuan1, LI Qiu-sha1, HAN Guo-zhu1, ZOU Ling-li1, LV Li1, ZHOU Qin1, LI Nan2 1. College of Pharmacy, Dalian Medical University, Dalian 116044, China 2. Department of Analytical Chemistry, Dalian University of Sciences and Technology, Dalian 116023, China.LC-MS/MS for Simultaneous Determination of Four Major Active Catechins of Tea Polyphenols in Rat Plasma and Its Application to Pharmacokinetics[J].Chinese Herbal Medicines,2010,2(4). 被引量：10
  • 3LI Qiu-sha1, ZOU Ling-li1, FU Ting1, HAN Guo-zhu1, L Li1, ZHOU Qin1, LIN Ya-fang1, LI Nan2 1. Department of Clinical Pharmacology, Dalian Medical University, Dalian 116044, China 2. Department of Analytical Chemistry, Dalian University of Science & Technology, Dalian 116023, China.Excretion of Four Catechins in Tea Polyphenols in Rats[J].Chinese Herbal Medicines,2009,1(1):59-65. 被引量：10
  • 4刘宏胜,戚爱棣.白藜芦醇抑制A172脑胶质瘤细胞生长的研究[J].药物评价研究,2009,32(2):121-122. 被引量：2
  • 5Yildirim M, Baysal V, Inaloz HS, et al. The role of oxidants and antioxidants in generalized vitiligo at tissue level ,IEADV, 2004, 18 : 683-686.
  • 6Hazneci E, Karabulut AB, Ozturk C, et al. A comparative study of superoxide dismutase, catalase, and glutathione peroxidase activities and nitrate levels in vitiligo patients. Int J Dermatol, 2005, 44: 636-640.
  • 7Beazley WD, Gaze D, Panske A, et aL Serum selenium levels and blood glutathione peroxidase activities in vitiligo. Br J Dermatol, 1999, 141:301-303.
  • 8Jimenez-cervantes C, Perez C. Inhibition of melanogenesis in response to oxidative stress : transient downregulation of melauocyte differentiation markers and possible involvement of microphthalmia transcription factor. J Cell Sci, 2001, 114: 2335-2344.
  • 9Agrawal D, Shajil EM, Mafratia YS, et al. Study on the antioxidant status of vitiligo patients of different age in Baroda. Piglnent Cell Res, 2004, 179 : 289-294.
  • 10Yildirim M, Baysal V, Inaloz HS, et al. The role of oxidants and antioxidants in generalized vitiligo at tissue level JEADV, 2004, 18 : 683-686.

共引文献11

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引证文献19

二级引证文献75

中国麻风皮肤病杂志
2016年 第3期

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